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indicated that canagliflozin could attenuate lipotoxicity and inflammatory injury in cardiomyocytes and protected diabetic mouse hearts via inhibiting the mTOR/hypoxia-inducible factor-1 (HIF-1) pathway [109]

Knowledge about cerebrovascular involvement in neurological disorders is rapidly expanding, and increasing evidence indicates that defects in angiogenesis, and the bloodbrain boundary are causally related to the cognitive dysfunctions that may be found in the course of neurodegenerative disorders (Nicolakakis and Hamel 2011

Study of literature revealed that deficiency of this vitamin occurs primarily due to insufficient dietary intake which results in a group of neurological symptoms in adults as well as infants

How GLP-1 Agonists Work Differently from GLP-1/Glucagon-like Peptide-1