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GLP-1R activity also promotes the transactivation of epidermal growth factor receptor (EGFR), which then signals through phosphoinositide 3-kinase (PI3K) and insulin receptor substrate-2 (IRS-2), and subsequently activates extracellular-signal-regulated kinase 1 and 2 (ERK1/2) and nuclear translocation of PKC to mediate -cell proliferation and differentiation (110, 114)

Studies have shown complex interactions between GHK-Cu and MMP-2, with effects that vary by concentration and cellular context

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This participation contributes to the amplification and persistence of low-grade chronic inflammation within the joint cavity ( Furthermore, the complex intercellular communication network within the joint, involving exosomes and various cytokines secreted by synovial cells or chondrocytes themselves, also participates in the fine-tuning of ferroptosis susceptibility