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In the WAG/Rij epilepsy rat model, a genetic model of absence epilepsy, epileptogenesis, and mild-depression comorbidity, the acetylated levels of H3 and H4 histones were increased significantly and the decreased expressions of HDAC1 and HDAC3 were observed following NaB treatment [132]

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The 3-NT, a marker of protein oxidative damage, is formed from peroxynitrite, a highly reactive free radical generated from nitric oxide (NO) and superoxide

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